Conference Description
The Mammalian DNA Repair GRS provides a unique forum for young doctoral and post-doctoral researchers to present their work, discuss new methods, cutting edge ideas, and pre-published data, as well as to build collaborative relationships with their peers. Experienced mentors and trainee moderators will facilitate active participation in scientific discussion to allow all attendees to be engaged participants rather than spectators.
Mammalian cells are constantly exposed to both endogenous and exogenous sources of DNA damaging agents which, if not resolved, can result in mutagenesis and genomic instability. Cells have developed specialized DNA repair pathways to identify and repair such damage. Dysregulation in these repair pathways is linked to various pathologies, including cancer and aging. In the upcoming Mammalian DNA Repair GRS, the central focus will be on unraveling the intricacies of DNA repair mechanisms in the context of DNA folding and chromatin organization. Oral and poster presentations will encourage discussion of recent and unpublished data in mammalian DNA repair. The program features a keynote seminar by a prominent expert in the field and includes a mentorship component, providing participants with opportunities to engage with panelists representing diverse scientific realms such as academia, industry, science communication, and publishing.
Application Instructions
The seminar will feature approximately 10 talks and 2 poster sessions. All attendees are expected to actively participate in the GRS, either by giving an oral presentation or presenting a poster. Therefore, all applications must include an abstract.
The seminar chair will select speakers from abstracts submitted by October 27, 2024. Those applicants who are not chosen for talks and those who apply after the deadline to be considered for an oral presentation will be expected to present a poster. In order to participate, you must submit an application by the date indicated in the Application Information section above.